![]() The parasite is also zoonotic, and cases of coenurosis have been reported in humans with coenuri located in the brain, spinal cord and eyes. However, coenurosis can also be controlled effectively through preventative measures, such as anthelmintic treatment of dogs and the proper disposal of intermediate host carcasses. Surgery is considered the most effective method for the treatment of cerebral coenurosis in small ruminants, but is often not carried out because of the limited finances of many sheep and goat breeders. ![]() Additionally, use of anthelmintics during the parasite’s migration stages reduces the development of cysts in the sheep brain. A vaccine based on recombinant oncosphere antigens has proved to be an effective tool against T. The best diagnostic method for cerebral coenurosis involves the interpretation of clinical signs with accurate localization of the cyst using diagnostic imaging techniques. Non-cerebral coenurosis has been most commonly reported in goats. Acute coenurosis occurs as a result of oncospheres migrating through the CNS, while chronic coenurosis occurs as a consequence of the coenurus maturing, which causes displacement and pressure atrophy of brain tissue. The disease mostly affects young sheep and is referred to as either acute or chronic coenurosis. multiceps populations has revealed significant genetic variation and the presence of three major haplotypes. Molecular characterization using the mitochondrial genes cytochrome c oxidase subunit 1 and nicotinamide adenine dinucleotide dehydrogenase subunit 1 of different T. multiceps has a worldwide distribution and is often fatal in intermediate hosts, which can result in substantial economic losses in livestock farming. Coenurosis caused by the larval stage of T. multiceps and its epidemiology, molecular characterization, pathogenesis, diagnosis, therapy, control and zoonotic potential. This review covers all aspects of the life cycle of T. The larval stage, Coenurus cerebralis, is typically found in the central nervous system (CNS) of a wide range of livestock and, to a lesser extent, in the extra-cerebral tissues of sheep and goats. Today, with various intervention programmes carried out by the gewog, the number of yak calves being infected with Gu-Yum is kept in check.Īpart from sensitizing people about vaccinating calves on time, the livestock extension office in Lunana carries out de-worming of dogs, and population control measures of wild dogs.Taenia multiceps is a taeniid cestode that inhabits the small intestines of both wild and domestic carnivores. ![]() In the past 15 years, over 9,500 yaks died of the gid in the country. We have to perform a surgery to remove the parasite,” Kinley Penjore said. But if they have ingested the parasite, it is very difficult to save them with medicines. Some even die in three months of infection. “If we are not able to give de-worming medicines and injections in time, some yaks live up to six months or a year only. If infected with gid and not treated on time, young calves will lose diet, suffer depression, paralysis, and will succumb to a slow, painful death. “These faeces are consumed by the yaks while grazing and gets infected with gid,” Kinley Penjor, the Livestock In-charge of Lunana, said In the process, the yaks are infected by the dogs’ faeces contaminated with the parasite. Highlanders use dogs to control and protect yaks from wild predators. Yak dogs among other canine species are considered as the main host for adult Gid tapeworm in the country. The larval stage of tapeworm Taenia multiceps infects the small intestine of carnivores. Gid is a disease of the central nervous system in yaks and sheep caused by Coenurus cerebralis. More than 10 yaks succumbed to the disease last year. The people of Lunana in Gasa lose their young yaks to Gid locally known as Gu-Yum disease every year.
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